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BMC Genomics 5, Weinger J. Why does a cell need to maintain its shape? What happens if we take away the cytoskeleton from What does cytoskeleton look like?
What cytoskeleton component is also used for motion? What functions of a cytoskeleton are unique to eukaryotic cells? See all questions in Cytoskeleton. Impact of this question views around the world.
These results identify for the first time a nontubulin component important for both the establishment and stabilization of the mitotic spindle apparatus in multicellular organisms.
We suggest that nonmicrotubule structural components may be important for these processes. Abstract The formation and maintenance of the bipolar mitotic spindle apparatus require a complex and balanced interplay of several mechanisms, including the stabilization and separation of polar microtubules and the action of various microtubule motors.
NuMA after 30 years: the matrix revisited. Requirements for NuMA in maintenance and establishment of mammalian spindle poles. Spindle assembly defects leading to the formation of a monopolar mitotic apparatus. The spindle function of CDCA4. Downregulation of Protein 4. Tankyrase function at telomeres, spindle poles, and beyond.
Inactivation of MAPK affects centrosome assembly, but not actin filament assembly, in mouse oocytes maturing in vitro. Cell and Molecular Biology of the Spindle Matrix. NuMA distribution and microtubule configuration in rabbit oocytes and cloned embryos.
Function of donor cell centrosome in intraspecies and interspecies nuclear transfer embryos. Association of the NuMA region on chromosome 11q13 with breast cancer susceptibility. NuMA in rat testis—Evidence for roles in proliferative activity and meiotic cell division. The first cell cycle after transfer of somatic cell nuclei in a non-human primate. NuMA expression and function in mouse oocytes and early embryos.
Functional role of centrosomes in spindle assembly and organization. NuMA and nuclear lamins are cleaved during viral infection—inhibition of caspase activity prevents cleavage and rescues HeLa cells from measles virus-induced but not from rhinovirus 1B-induced cell death.
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